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In the human body, carnitine is mainly found in muscles and as muscles are unable to synthesize carnitine they rely on an active transport across the sarcolemma to provide L-carnitine to muscle cells

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While current studies have delineated the core roles of ferroptosis (via the System Xc GSHGPX4 axis, iron overload, lipid peroxidation, and mitochondrial dysfunction) and identified antioxidant networks (Nrf2/Sirtuins/FTH1) as therapeutic targets in DN, several critical research gaps persist that hinder a comprehensive understanding of ferroptosis-driven DN progression and translational application

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